Bengi Ünal is a PostDoc in the Disorders of Neural Circuit Function research group at Max Planck Florida Institute for Neuroscience.
BENGI ÜNAL, PHD
Dr. Bengi Ünal joined MPFI in 2013 where she studies neural circuit changes in autism spectrum disorders. Prior to joining the Bolton Lab, Bengi's research included dissecting the basal ganglia macrocircuitry in Huntington's disease. She received her PhD in Behavioral and Neural Sciences from Rutgers University in 2012. As a graduate student, Bengi conducted in vitro electrophysiological and anatomical experiments investigating the changes in tyrosine hydroxylase-expressing interneurons following striatal dopamine denervation.
- Ünal B, Shah F, Kothari J, Tepper JM (2014) Anatomical and electrophysiological changes in striatal tyrosine hydroxylase-expressing interneurons after nigrostriatal dopamine loss. Brain Structure and Function. In Press.
- Kamal A, Ramakers GM, Ünal B, Kas MJ (2014) Social isolation stress reduces hippocampal long-term potentiation: effect of animal strain and involvement of glucocorticoid receptors. Neuroscience, 256: 262-70.
- Ibanez-Sandoval O, Tecuapetla F, Ünal B, Shah F, Koos T, Tepper JM (2011) A novel functionally distinct striatal neuropeptide Y interneuron. Journal of Neuroscience, 31(46): 16757-16769.
- Ünal B, Ibanez-Sandoval O, Shah F, Abercrombie, ED, Tepper JM (2011) Distribution of striatal tyrosine-hydroxylase expressing interneurons with respect to anatomical organization of the striatum. Frontiers in Neuroscience, 5:41.
- Ibanez-Sandoval O, Tecuapetla F, Ünal B, Shah F, Tepper JM (2010) Electrophysiological and morphological characteristicss and synaptic connectivity of tyrosine hydroxylase- expressing neurons in adult mice striatum. Journal of Neuroscience, 30(20): 6999-7016.
- Ünal B and Manahan-Vaughan D. (2009) A specific role for group II metabotropic glutamate receptors in hippocampal long-term depression and spatial memory. Neuroscience, 158(1): 149-58.
- Ünal B and Manahan-Vaughan D. (2007). Antagonism of group III metabotropic glutamate receptors results in impairment of LTD but not LTP in the hippocampal CA1 region, and prevents long-term spatial memory. European Journal of Neuroscience, 26(5): 1166-72.